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MUSCLE GROWTH AND FAT APOPTOSIS- MECHANISM OF ACTION
TeslaSculpt uses high intensity electromagnetic technology, which has the ability to induce supramaximal muscle contractions.
The Rapidly Chaning Magnetic Flield induces electric waves in the tissue where it polarizes neural membranes and governs motor units in the target muscle tissue, causing concentric contractions,
During treatment, the muscles are contracted to supramaximal
levels, which is forced to adapt to such extreme condition.
Intensive supramaximal contractions enhance release of epinephrine
which triggers a cascade effect leading to supramaximal lipolysis
in fat cells. Because the stimulation is so rapid and intensive,
the lipolytic response is excessive and FFAs start
over-accumulating in dipocytes. The FFA overflow causes cell
dysfunction and induction of apoptosis – programmed cell death.
Dead cells collapse and are naturally flushed away.
Meanwhile, the musle responds with a deep remodeling of its inner
structure, induces the growth of myofibrils (muscle hypertrophy),
the creation of new protein strands and the growth of muscle fibers
(muscle hyperplasia). The process results in increased muscle
density and volume.
The effects on fat tissue
During Teslasculpt treatment, Supramaximal muscle contractions
induce an extreme catabolic reaction and supramaximal lipolysis to
offer energy source for the needed muscle activity and body
metabolism, which in turn brings about a dramatic release of FFA
When the amount of released FFA exceed normal levels, they start
accumulating intracellularly in surrounding adipocytes and
eventually lead to their dysfunction.
This catabolic and supramaximal lipolysis effect occurs mostly in
the area in close proximity to the actual contracting muscles, due
to increased adipose tissue blood flow andparacrine substances
released from the contracting muscles.
The effects on muscle tissue
When exposed to supramaximal contractions, the muscle tissue is forced to adapt to such extreme conditions and responds with a deep remodeling of its inner structure, i.e., the growth of myofibrils (muscle hypertrophy) and creation of new protein strands and muscle fibers (muscle hyperplasia).5-7 Increased muscle